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• W4313361255 startingPage "75" @default.
• W4313361255 abstract "In diseases with concomitant oxidative stress, chronic multi-hormonal resistances could be detected. The most conspicuous component of these resistances is insulin resistance, but also leptin, erythropoietin, acetylcholine, triiodothyronine and glucagon-like peptide-1 resistances also occur. On the other hand, in oxidative stress, abnormal tyrosines, for instance, meta- and ortho-tyrosine are also produced and incorporated into the proteins through the translational process. In case these modified proteins are components of the intracellular signalling pathways, a hormonal resistance may develop. The above-mentioned hormones, owning overlapping signalling pathways at the insulin receptor substrate, develop an abnormal tyrosine phosphorylation dependent chronic multi-hormonal resistance. A few weeks free of oxidative stress or the use of antioxidant therapy are required to provide a return from this resistance, which return may be further supported by the supplementation of physiological para-tyrosine and by the add-on therapy of a pharmacological dose of glucagon-like peptide-1 receptor agonist, which is able to bypass the critical insulin receptor substrate signalling." @default.
• W4313361255 created "2023-01-06" @default.
• W4313361255 creator A5028717346 @default.
• W4313361255 date "2022-12-29" @default.
• W4313361255 modified "2023-09-30" @default.
• W4313361255 title "The Common Single Cause of Chronic Multi-Hormonal Resistance in Oxidative Stress" @default.
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• W4313361255 doi "https://doi.org/10.3390/antiox12010075" @default.
• W4313361255 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/36670938" @default.
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