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- W4313374040 abstract "Epithelial–mesenchymal transition (EMT), a cellular phenotypic change from epithelial to mesenchymal-like features, is related to the resistance and metastasis of cancer stem cells (CSCs). Several signal transduction mechanisms induce EMT, which causes the gene expression alteration to induce the acquisition of resistance and metastasis in cancer. EMT is characterized with high gene expression of cadherin 2 (N-cadherin) and vimentin, and sparse cell–cell junction. The cells with EMT-phenotype have migration, metastasis and drug-resistance capacity, which are main characteristics of CSCs. It seems that the main population of CSCs exhibits EMT phenotype, whereas some populations consist of phenotypes other than EMT. In this chapter, EMT mechanism, phenotypic features of EMT and CSCs, signal transduction in EMT and CSCs, differences between EMT and CSCs, and the role of EMT in CSCs are described." @default.
- W4313374040 created "2023-01-06" @default.
- W4313374040 creator A5085406316 @default.
- W4313374040 date "2022-01-01" @default.
- W4313374040 modified "2023-10-16" @default.
- W4313374040 title "Epithelial–Mesenchymal Transition and Cancer Stem Cells" @default.
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- W4313374040 doi "https://doi.org/10.1007/978-3-031-12974-2_1" @default.
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