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- W4313388301 abstract "Abstract B cells are implicated in the pathogenesis of multiple sclerosis. Data from the Experimental Autoimmune Encephalomyelitis (EAE) model are contradictory. We previously showed that myelin oligodendrocyte glycoprotein (rMOG)-induced EAE was B cell-dependent, while MOG35-55-induced disease was B cell-independent. Differences in the intrathecal B cell population may be relevant to the role of B cells in these two models. Higher numbers of B cells were detected in the CNS of rMOG-primed mice compared to MOG35-55-primed mice. Analysis of chemokine receptor expression by B cells isolated from the CNS of rMOG- and MOG35-55-primed mice was performed to further characterize the recruitment of B cells to the CNS during disease. Differences in the expression of CXCR1 and CCR4 was initially suggested by RT-PCR and confirmed by Q-PCR. Preliminary analysis of the cognate chemokines for these receptors indicated differences in the absolute expression as well as the expression profile of these ligands. Thus, differences in expression of factors important in B cell migration in rMOG- and MOG35-55-induced EAE may be relevant to the recruitment of pathogenic B cells in B cell-dependent EAE." @default.
- W4313388301 created "2023-01-06" @default.
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- W4313388301 date "2007-04-01" @default.
- W4313388301 modified "2023-09-27" @default.
- W4313388301 title "Differences in the expression of chemokine receptors by intrathecal B cells in B cell-dependent vs. B cell-independent EAE (96.25)" @default.
- W4313388301 doi "https://doi.org/10.4049/jimmunol.178.supp.96.25" @default.
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