Matches in SemOpenAlex for { <https://semopenalex.org/work/W4313488278> ?p ?o ?g. }
Showing items 1 to 52 of
52
with 100 items per page.
- W4313488278 endingPage "912" @default.
- W4313488278 startingPage "903" @default.
- W4313488278 abstract "Vascular calcification is a common pathological process in patients with diabetes, chronic kidney disease, and cardiovascular disease, manifested by the deposition of hydroxyapatite on the walls of blood vessels. Hydrogen sulfide is the third gas signal molecule found in mammals after nitric oxide and carbon monoxide, which has anti-inflammatory, antioxidant stress and other effects in the cardiovascular system. In recent years, it has been recognized that hydrogen sulfide has an anti-vascular calcification effect, and supplementation with hydrogen sulfide and its donors can alleviate vascular calcification. In this review, we discussed the various evidence of the protective effect of hydrogen sulfide on vascular calcification, and highlighted the hydrogen sulfide metabolism changes and the potential regulatory mechanisms of hydrogen sulfide on the pathophysiological changes in vascular calcification." @default.
- W4313488278 created "2023-01-06" @default.
- W4313488278 creator A5010479652 @default.
- W4313488278 creator A5019213073 @default.
- W4313488278 creator A5037181329 @default.
- W4313488278 date "2022-12-25" @default.
- W4313488278 modified "2023-09-30" @default.
- W4313488278 title "[Role and mechanism of hydrogen sulfide in vascular calcification]." @default.
- W4313488278 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/36594379" @default.
- W4313488278 hasPublicationYear "2022" @default.
- W4313488278 type Work @default.
- W4313488278 citedByCount "0" @default.
- W4313488278 crossrefType "journal-article" @default.
- W4313488278 hasAuthorship W4313488278A5010479652 @default.
- W4313488278 hasAuthorship W4313488278A5019213073 @default.
- W4313488278 hasAuthorship W4313488278A5037181329 @default.
- W4313488278 hasConcept C126322002 @default.
- W4313488278 hasConcept C178790620 @default.
- W4313488278 hasConcept C185592680 @default.
- W4313488278 hasConcept C2780309369 @default.
- W4313488278 hasConcept C2780564542 @default.
- W4313488278 hasConcept C518881349 @default.
- W4313488278 hasConcept C519581460 @default.
- W4313488278 hasConcept C71924100 @default.
- W4313488278 hasConceptScore W4313488278C126322002 @default.
- W4313488278 hasConceptScore W4313488278C178790620 @default.
- W4313488278 hasConceptScore W4313488278C185592680 @default.
- W4313488278 hasConceptScore W4313488278C2780309369 @default.
- W4313488278 hasConceptScore W4313488278C2780564542 @default.
- W4313488278 hasConceptScore W4313488278C518881349 @default.
- W4313488278 hasConceptScore W4313488278C519581460 @default.
- W4313488278 hasConceptScore W4313488278C71924100 @default.
- W4313488278 hasIssue "6" @default.
- W4313488278 hasLocation W43134882781 @default.
- W4313488278 hasOpenAccess W4313488278 @default.
- W4313488278 hasPrimaryLocation W43134882781 @default.
- W4313488278 hasRelatedWork W145579800 @default.
- W4313488278 hasRelatedWork W1970132168 @default.
- W4313488278 hasRelatedWork W2023776203 @default.
- W4313488278 hasRelatedWork W2045821258 @default.
- W4313488278 hasRelatedWork W2059344404 @default.
- W4313488278 hasRelatedWork W2398114610 @default.
- W4313488278 hasRelatedWork W2748952813 @default.
- W4313488278 hasRelatedWork W2899084033 @default.
- W4313488278 hasRelatedWork W2950894205 @default.
- W4313488278 hasRelatedWork W1664655889 @default.
- W4313488278 hasVolume "74" @default.
- W4313488278 isParatext "false" @default.
- W4313488278 isRetracted "false" @default.
- W4313488278 workType "article" @default.