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- W4313492025 abstract "Apolipoprotein M (ApoM) binds sphingosine-1-phosphate (S1P) and is inversely associated with mortality in human heart failure (HF). Here, we show that anthracyclines such as doxorubicin (Dox) reduce circulating ApoM in mice and humans, that ApoM is inversely associated with mortality in patients with anthracycline-induced heart failure, and ApoM heterozygosity in mice increases Dox-induced mortality. In the setting of Dox stress, our studies suggest ApoM can help sustain myocardial autophagic flux in a post-transcriptional manner, attenuate Dox cardiotoxicity, and prevent lysosomal injury." @default.
- W4313492025 created "2023-01-06" @default.
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- W4313492025 date "2023-03-01" @default.
- W4313492025 modified "2023-10-14" @default.
- W4313492025 title "Apolipoprotein M Attenuates Anthracycline Cardiotoxicity and Lysosomal Injury" @default.
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- W4313492025 doi "https://doi.org/10.1016/j.jacbts.2022.09.010" @default.
- W4313492025 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/37034289" @default.
- W4313492025 hasPublicationYear "2023" @default.