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- W4313531321 endingPage "114199" @default.
- W4313531321 startingPage "114199" @default.
- W4313531321 abstract "As the final product of purine metabolism, excess serum uric acid (SUA) aggravates the process of some metabolic diseases. SUA causes renal tubule damage, interstitial fibrosis, and glomerular hardening, leading to gouty nephropathy (GN). A growing number of investigations have shown that NF-κB mediated inflammation and oxidative stress have been directly involved in the pathogenesis of GN. Traditional Chinese medicine's treatment methods of GN have amassed a wealth of treatment experience. In this review, we first describe the mechanism of NF-κB signaling pathways in GN. Subsequently, we highlight traditional Chinese medicine that can treat GN through NF-κB pathways. Finally, commenting on promising candidate targets of herbal medicine for GN treatment via suppressing NF-κB signaling pathways was summarized." @default.
- W4313531321 created "2023-01-06" @default.
- W4313531321 creator A5003642180 @default.
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- W4313531321 creator A5065201117 @default.
- W4313531321 creator A5073070877 @default.
- W4313531321 creator A5087929908 @default.
- W4313531321 date "2023-02-01" @default.
- W4313531321 modified "2023-10-17" @default.
- W4313531321 title "Therapeutic effects of traditional Chinese medicine on gouty nephropathy: Based on NF-κB signalingpathways" @default.
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