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- W4313593541 endingPage "112647" @default.
- W4313593541 startingPage "112647" @default.
- W4313593541 abstract "UV-A radiation affects skin homeostasis by promoting oxidative distress. Endogenous photosensitizers in the dermis and epidermis of human skin absorb UV-A radiation forming excited states (singlet and triplet) and reactive oxygen species (ROS) producing oxidized compounds that trigger biological responses. The activation of NF-kB induces the expression of pro-inflammatory cytokines and can intensify the generation of ROS. However, there is no studies evaluating the cross talks between inflammatory stimulus and UV-A exposure on the levels of redox misbalance and inflammation. In here, we evaluated the effects of UV-A exposure on J774 macrophage cells previously challenged with LPS in terms of oxidative distress, release of pro-inflammatory cytokines, and activation of regulated cell death pathways. Our results showed that LPS potentiates the dose-dependent UV-A-induced oxidative distress and cytokine release, in addition to amplifying the regulated (autophagy and apoptosis) and non-regulated (necrosis) mechanisms of cell death, indicating that a previous inflammatory stimulus potentiates UV-A-induced cell damage. We discuss these results in terms of the current-available skin care strategies." @default.
- W4313593541 created "2023-01-06" @default.
- W4313593541 creator A5012172291 @default.
- W4313593541 creator A5014963747 @default.
- W4313593541 creator A5024456761 @default.
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- W4313593541 creator A5067356145 @default.
- W4313593541 creator A5080188187 @default.
- W4313593541 creator A5083448992 @default.
- W4313593541 creator A5084538695 @default.
- W4313593541 date "2023-02-01" @default.
- W4313593541 modified "2023-09-27" @default.
- W4313593541 title "Inflammatory stimulus worsens the effects of UV-A exposure on J774 cells" @default.
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- W4313593541 doi "https://doi.org/10.1016/j.jphotobiol.2023.112647" @default.
- W4313593541 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/36634432" @default.
- W4313593541 hasPublicationYear "2023" @default.
- W4313593541 type Work @default.