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- W4315705329 abstract "We thank M. Ackermann and co-workers for their interest in our recent publication [1]. These authors highlight our report of persistent increased alveolar dead space in 30% of 17 patients studied within ∼2 months after an acute episode of mild–moderate COVID-19. After outlining their own work, demonstrating secondary pulmonary lobule pathologies found in the lungs of deceased COVID-19 patients, they then hypothesise that secondary lobular micro-ischaemia may be responsible for the elevated alveolar dead space found in our study. Increased dead space following COVID-19 may be due to microvascular injury or secondary micro-ischaemia https://bit.ly/3Fypdwz" @default.
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- W4315705329 date "2023-01-12" @default.
- W4315705329 modified "2023-09-30" @default.
- W4315705329 title "Reply: Intrapulmonary shunt and alveolar dead space in a cohort of patients with acute COVID-19 pneumonitis and early recovery" @default.
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- W4315705329 doi "https://doi.org/10.1183/13993003.02287-2022" @default.
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