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- W4316672739 endingPage "1801" @default.
- W4316672739 startingPage "1801" @default.
- W4316672739 abstract "Myofibroblasts escape apoptosis and proliferate abnormally under pathological conditions, especially fibrosis; they synthesize and secrete a large amount of extracellular matrix (ECM), such as α-SMA and collagen, which leads to the distortion of organ parenchyma structure, an imbalance in collagen deposition and degradation, and the replacement of parenchymal cells by fibrous connective tissues. Fibroblast to myofibroblast transition (FMT) is considered to be the main source of myofibroblasts. Therefore, it is crucial to explore the influencing factors regulating the process of FMT for the prevention, treatment, and diagnosis of FMT-related diseases. In recent years, non-coding RNAs, including microRNA, long non-coding RNAs, and circular RNAs, have attracted extensive attention from scientists due to their powerful regulatory functions, and they have been found to play a vital role in regulating FMT. In this review, we summarized ncRNAs which regulate FMT during fibrosis and found that they mainly regulated signaling pathways, including TGF-β/Smad, MAPK/P38/ERK/JNK, PI3K/AKT, and WNT/β-catenin. Furthermore, the expression of downstream transcription factors can be promoted or inhibited, indicating that ncRNAs have the potential to be a new therapeutic target for FMT-related diseases." @default.
- W4316672739 created "2023-01-17" @default.
- W4316672739 creator A5012140765 @default.
- W4316672739 creator A5014329370 @default.
- W4316672739 creator A5062933461 @default.
- W4316672739 creator A5075877965 @default.
- W4316672739 date "2023-01-16" @default.
- W4316672739 modified "2023-09-25" @default.
- W4316672739 title "Noncoding RNAs: Master Regulator of Fibroblast to Myofibroblast Transition in Fibrosis" @default.
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