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W4319441023 endingPage "530" @default.
W4319441023 startingPage "530" @default.
W4319441023 abstract "The application of immunotherapy for cancer treatment is rapidly becoming more widespread. Immunotherapeutic agents are frequently combined with various types of treatments to obtain a more durable antitumor clinical response in patients who have developed resistance to monotherapy. Chemotherapeutic drugs that induce DNA damage and trigger DNA damage response (DDR) frequently induce an increase in the expression of the programmed death ligand-1 (PD-L1) that can be employed by cancer cells to avoid immune surveillance. PD-L1 exposed on cancer cells can in turn be targeted to re-establish the immune-reactive tumor microenvironment, which ultimately increases the tumor's susceptibility to combined therapies. Here we review the recent advances in how the DDR regulates PD-L1 expression and point out the effect of etoposide, irinotecan, and platinum compounds on the anti-tumor immune response." @default.
W4319441023 created "2023-02-09" @default.
W4319441023 creator A5008965047 @default.
W4319441023 creator A5016582690 @default.
W4319441023 creator A5023057502 @default.
W4319441023 creator A5025317264 @default.
W4319441023 creator A5032027594 @default.
W4319441023 creator A5067813322 @default.
W4319441023 creator A5079629568 @default.
W4319441023 date "2023-02-07" @default.
W4319441023 modified "2023-10-18" @default.
W4319441023 title "PD-1/PD-L1 and DNA Damage Response in Cancer" @default.
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