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- W4320000318 abstract "Thyroid hormones activate nuclear receptors to alter the transcription of a range of major cardiac proteins, thus affecting numerous cardiac functions. Overt hyperthyroidism decreases systolic vascular resistance, increases cardiac contractility and heart rate, and is associated with systemic and pulmonary hypertension, especially systolic hypertension. Overt hypothyroidism has the opposite effect to increase systemic vascular resistance, decrease cardiac contractility, and increase diastolic blood pressure. Impairment of renal perfusion in hypothyroidism may also contribute to the hypertensive effect of hypothyroidism. Treatment improves systemic and pulmonary hypertension. The effects of both subclinical hyperthyroidism and subclinical hypothyroidism are less uniform. Primary hyperparathyroidism is associated with hypertension. Hypercalcemia increases vascular contractility, impairs vasodilatation, and increases systemic vascular resistance, but the elevation of parathyroid hormone level may also have a direct effect. The cardiovascular benefit of treatment of hyperparathyroidism is more inconsistent." @default.
- W4320000318 created "2023-02-11" @default.
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- W4320000318 date "2023-01-01" @default.
- W4320000318 modified "2023-09-27" @default.
- W4320000318 title "Hypertension in thyroid disease and primary hyperparathyroidism" @default.
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- W4320000318 doi "https://doi.org/10.1016/b978-0-323-96120-2.00005-4" @default.
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