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- W4321523333 abstract "Across the world a dementia case is diagnosed every three seconds. Alzheimer’s disease (AD) causes 50–60% of these cases. The most prominent theory for AD correlates the deposition of amyloid beta (Aβ) with the onset of dementia. Whether Aβ is causative remains unclear due to findings such as the recently approved drug Aducanumab showing effective clearance of Aβ, but not improving cognition. New approaches for understanding Aβ function, are therefore necessary. Here we discuss the application of optogenetic techniques to gain insight into AD. Optogenetics, or genetically encoded, light-dependent on/off switches, provides precise spatiotemporal control to regulate cellular dynamics. This precise control over protein expression and oligomerization or aggregation could provide a better understanding of the etiology of AD." @default.
- W4321523333 created "2023-02-24" @default.
- W4321523333 creator A5035852093 @default.
- W4321523333 creator A5058460183 @default.
- W4321523333 date "2023-02-21" @default.
- W4321523333 modified "2023-10-14" @default.
- W4321523333 title "Using Optogenetics to Model Cellular Effects of Alzheimer’s Disease" @default.
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- W4321523333 doi "https://doi.org/10.3390/ijms24054300" @default.
- W4321523333 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/36901729" @default.
- W4321523333 hasPublicationYear "2023" @default.
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