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- W4321638862 abstract "Abstract The malaria parasite Plasmodium falciparum causes substantial human mortality, primarily in equatorial Africa. Enriched in affected African populations, the B*53 variant of HLA-B, a cell surface protein that presents peptide antigens to cytotoxic lymphocytes, confers protection against severe malaria. Gorilla, chimpanzee, and bonobo are humans’ closest living relatives. These African apes have HLA-B orthologs and are infected by parasites in the same subgenus ( Laverania ) as P. falciparum , but the consequences of these infections are unclear. Laverania parasites infect bonobos ( Pan paniscus ) at only one (TL2) of many sites sampled across their range. TL2 spans the Lomami River and has genetically divergent subpopulations of bonobos on each side. Papa-B, the bonobo ortholog of HLA-B, includes variants having a B*53-like (B07) peptide-binding supertype profile. Here we show that B07 Papa-B occur at high frequency in TL2 bonobos and that malaria appears to have independently selected for different B07 alleles in the two subpopulations." @default.
- W4321638862 created "2023-02-24" @default.
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- W4321638862 date "2023-02-23" @default.
- W4321638862 modified "2023-10-18" @default.
- W4321638862 title "Malaria-driven adaptation of MHC class I in wild bonobo populations" @default.
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- W4321638862 doi "https://doi.org/10.1038/s41467-023-36623-9" @default.
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- W4321638862 hasPublicationYear "2023" @default.
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