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- W4322740902 endingPage "605" @default.
- W4322740902 startingPage "605" @default.
- W4322740902 abstract "The increase in environmental temperature due to global warming is a critical threat to plant growth and productivity. Heat stress can cause impairment in several biochemical and physiological processes. Plants sense and respond to this adverse environmental condition by activating a plethora of defense systems. Among them, the heat stress response (HSR) involves an intricate network of heat shock factors (HSFs) and heat shock proteins (HSPs). However, a growing amount of evidence suggests that reactive oxygen species (ROS), besides potentially being responsible for cellular oxidative damage, can act as signal molecules in HSR, leading to adaptative responses. The role of ROS as toxic or signal molecules depends on the fine balance between their production and scavenging. Enzymatic and non-enzymatic antioxidants represent the first line of defense against oxidative damage and their activity is critical to maintaining an optimal redox environment. However, the HS-dependent ROS burst temporarily oxidizes the cellular environment, triggering redox-dependent signaling cascades. This review provides an overview of the redox-activated mechanisms that participate in the HSR." @default.
- W4322740902 created "2023-03-03" @default.
- W4322740902 creator A5001167706 @default.
- W4322740902 creator A5005426454 @default.
- W4322740902 creator A5017767747 @default.
- W4322740902 creator A5031875297 @default.
- W4322740902 creator A5071977086 @default.
- W4322740902 date "2023-03-01" @default.
- W4322740902 modified "2023-10-16" @default.
- W4322740902 title "Redox Signaling in Plant Heat Stress Response" @default.
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