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- W4324134947 startingPage "112237" @default.
- W4324134947 abstract "Transitions in competence underlie the ability of CNS progenitors to generate a diversity of neurons and glia. Retinal progenitor cells in mouse generate early-born cell types embryonically and late-born cell types largely postnatally. We find that the transition from early to late progenitor competence is regulated by Jarid2. Loss of Jarid2 results in extended production of early cell types and extended expression of early progenitor genes. Jarid2 can regulate histone modifications, and we find reduction of repressive mark H3K27me3 on a subset of early progenitor genes with loss of Jarid2, most notably Foxp1. We show that Foxp1 regulates the competence to generate early-born retinal cell types, promotes early and represses late progenitor gene expression, and is required for extending early retinal cell production after loss of Jarid2. We conclude that Jarid2 facilitates progression of retinal progenitor temporal identity by repressing Foxp1, which is a primary regulator of early temporal patterning." @default.
- W4324134947 created "2023-03-15" @default.
- W4324134947 creator A5004218480 @default.
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- W4324134947 creator A5043522586 @default.
- W4324134947 creator A5053278255 @default.
- W4324134947 creator A5079156452 @default.
- W4324134947 date "2023-03-01" @default.
- W4324134947 modified "2023-10-14" @default.
- W4324134947 title "Jarid2 promotes temporal progression of retinal progenitors via repression of Foxp1" @default.
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- W4324134947 doi "https://doi.org/10.1016/j.celrep.2023.112237" @default.
- W4324134947 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/36924502" @default.