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• W4324325002 abstract "HomeHypertensionVol. 80, No. 4Phenotype-Specific Induced Pluripotent Stem Cell–Derived Vascular Smooth Muscle Cells to Model Vascular Disease: Implications of Differentiation Protocols No AccessEditorialRequest AccessFull TextAboutView Full TextView PDFView EPUBSections ToolsAdd to favoritesDownload citationsTrack citationsPermissions ShareShare onFacebookTwitterLinked InMendeleyReddit Jump toNo AccessEditorialRequest AccessFull TextPhenotype-Specific Induced Pluripotent Stem Cell–Derived Vascular Smooth Muscle Cells to Model Vascular Disease: Implications of Differentiation Protocols Livia L. Camargo and Rhian M. Touyz Livia L. CamargoLivia L. Camargo Correspondence to: Livia L. Camargo, PhD, Research Institute of the McGill University Health Centre, McGill University, Site Glen Block E, Bureau/Office E01.3362, 1001 Decarie Blvd, Montreal, Quebec H4A3J1, Canada. Email E-mail Address: [email protected] https://orcid.org/0000-0002-7451-7147 Research Institute of the McGill University Health Centre, McGill University, Montreal, Quebec, Canada. Search for more papers by this author and Rhian M. TouyzRhian M. Touyz https://orcid.org/0000-0003-0670-0887 Research Institute of the McGill University Health Centre, McGill University, Montreal, Quebec, Canada. Search for more papers by this author Originally published15 Mar 2023https://doi.org/10.1161/HYPERTENSIONAHA.123.20871Hypertension. 2023;80:754–756This article is a commentary on the followingGenomic, Transcriptomic, and Proteomic Depiction of Induced Pluripotent Stem Cells–Derived Smooth Muscle Cells As Emerging Cellular Models for Arterial DiseasesFootnotesFor Sources of Funding and Disclosures, see page 756.The opinions expressed in this article are not necessarily those of the editors nor the American Heart Association.Correspondence to: Livia L. Camargo, PhD, Research Institute of the McGill University Health Centre, McGill University, Site Glen Block E, Bureau/Office E01.3362, 1001 Decarie Blvd, Montreal, Quebec H4A3J1, Canada. Email livia.lucca@rimuhc.caReferences1. Owens GK, Kumar MS, Wamhoff BR. Molecular regulation of vascular smooth muscle cell differentiation in development and disease.Physiol Rev. 2004; 84:767–801. doi: 10.1152/physrev.00041.2003CrossrefMedlineGoogle Scholar2. Chakraborty R, Chatterjee P, Dave JM, Ostriker AC, Greif DM, Rzucidlo EM, Martin KA. Targeting smooth muscle cell phenotypic switching in vascular disease.JVS Vasc Sci. 2021; 2:79–94. doi: 10.1016/j.jvssci.2021.04.001CrossrefMedlineGoogle Scholar3. Liu L, Jouve C, Henry J, Berrandou TE, Hulot JS, Georges A, Bouatia-Naji N. Genomic, transcriptomic, and proteomic depiction of induced pluripotent stem cell–derived smooth muscle cells as emerging cellular models for arterial diseases.Hypertension. 2023; 80:740–753. doi: 10.1161/HYPERTENSIONAHA.122.19733LinkGoogle Scholar4. Liu M, Gomez D. Smooth muscle cell phenotypic diversity.Arterioscler Thromb Vasc Biol. 2019; 39:1715–1723. doi: 10.1161/atvbaha.119.312131LinkGoogle Scholar5. Shen M, Quertermous T, Fischbein MP, Wu JC. Generation of vascular smooth muscle cells from induced pluripotent stem cells: methods, applications, and considerations.Circ Res. 2021; 128:670–686. doi: 10.1161/circresaha.120.318049LinkGoogle Scholar6. Tang Y, Urs S, Boucher J, Bernaiche T, Venkatesh D, Spicer DB, Vary CP, Liaw L. Notch and transforming growth factor-beta (TGFbeta) signaling pathways cooperatively regulate vascular smooth muscle cell differentiation.J Biol Chem. 2010; 285:17556–17563. doi: 10.1074/jbc.M109.076414CrossrefMedlineGoogle Scholar7. Thomas JA, Deaton RA, Hastings NE, Shang Y, Moehle CW, Eriksson U, Topouzis S, Wamhoff BR, Blackman BR, Owens GK. PDGF-DD, a novel mediator of smooth muscle cell phenotypic modulation, is upregulated in endothelial cells exposed to atherosclerosis-prone flow patterns.Am J Physiol Heart Circ Physiol. 2009; 296:H442–H452. doi: 10.1152/ajpheart.00165.2008CrossrefMedlineGoogle Scholar8. Zhang J, McIntosh BE, Wang B, Brown ME, Probasco MD, Webster S, Duffin B, Zhou Y, Guo LW, Burlingham WJ, et al. A human pluripotent stem cell-based screen for smooth muscle cell differentiation and maturation identifies inhibitors of intimal hyperplasia.Stem Cell Rep. 2019; 12:1269–1281. doi: 10.1016/j.stemcr.2019.04.013CrossrefMedlineGoogle Scholar9. Bravo DD, Chernov-Rogan T, Chen J, Wang J. An impedance-based cell contraction assay using human primary smooth muscle cells and fibroblasts.J Pharmacol Toxicol Methods. 2018; 89:47–53. doi: 10.1016/j.vascn.2017.10.006CrossrefMedlineGoogle Scholar10. Camargo LL, Harvey AP, Rios FJ, Tsiropoulou S, Da Silva RNO, Cao Z, Graham D, McMaster C, Burchmore RJ, Hartley RC, et al. Vascular Nox (NADPH Oxidase) compartmentalization, protein hyperoxidation, and endoplasmic reticulum stress response in hypertension.Hypertension. 2018; 72:235–246. doi: 10.1161/HYPERTENSIONAHA.118.10824LinkGoogle Scholar11. Chattopadhyay A, Kwartler CS, Kaw K, Li Y, Kaw A, Chen J, LeMaire SA, Shen YH, Milewicz DM. Cholesterol-induced phenotypic modulation of smooth muscle cells to macrophage/fibroblast-like cells is driven by an unfolded protein response.Arterioscler Thromb Vasc Biol. 2021; 41:302–316. doi: 10.1161/ATVBAHA.120.315164. Epub 2020 Oct 8LinkGoogle Scholar12. da Silva RA, da S Feltran G, da C Fernandes CJ, Zambuzzi WF. Osteogenic gene markers are epigenetically reprogrammed during contractile-to-calcifying vascular smooth muscle cell phenotype transition.Cell Signal. 2020; 66:109458. doi: 10.1016/j.cellsig.2019.109458CrossrefMedlineGoogle Scholar eLetters(0)eLetters should relate to an article recently published in the journal and are not a forum for providing unpublished data. Comments are reviewed for appropriate use of tone and language. Comments are not peer-reviewed. Acceptable comments are posted to the journal website only. Comments are not published in an issue and are not indexed in PubMed. Comments should be no longer than 500 words and will only be posted online. References are limited to 10. Authors of the article cited in the comment will be invited to reply, as appropriate.Comments and feedback on AHA/ASA Scientific Statements and Guidelines should be directed to the AHA/ASA Manuscript Oversight Committee via its Correspondence page.Sign In to Submit a Response to This Article Previous Back to top Next FiguresReferencesRelatedDetailsRelated articlesGenomic, Transcriptomic, and Proteomic Depiction of Induced Pluripotent Stem Cells–Derived Smooth Muscle Cells As Emerging Cellular Models for Arterial DiseasesLu Liu, et al. Hypertension. 2023;80:740-753 April 2023Vol 80, Issue 4 Advertisement Article InformationMetrics © 2023 American Heart Association, Inc.https://doi.org/10.1161/HYPERTENSIONAHA.123.20871PMID: 36921028 Originally publishedMarch 15, 2023 PDF download Advertisement SubjectsContractile FunctionSmooth Muscle Proliferation and DifferentiationStem CellsVascular Biology" @default.
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