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- W4328012208 abstract "Background: Acute myeloid leukemia (AML) remains a highly fatal disease. Based on the available evidence of deregulated pathways of proliferation and apoptosis in AML, we hypothesize that pharmacological modulation of AML blasts by inhibitors of BCL-2 (Venetoclax) and novel CDK7 inhibitor (CRI-256) will be potently synergistic for the treatment of AML." @default.
- W4328012208 created "2023-03-22" @default.
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- W4328012208 creator A5087775526 @default.
- W4328012208 date "2022-08-01" @default.
- W4328012208 modified "2023-09-25" @default.
- W4328012208 title "Novel CDK-7 Inhibitor Suppresses Transcription of Oncogenes and Enhances Venetoclax-Mediated Apoptosis in Acute Myeloid Leukemia" @default.
- W4328012208 doi "https://doi.org/10.1055/s-0042-1755486" @default.
- W4328012208 hasPublicationYear "2022" @default.
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