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- W43439608 abstract "We have sought to exploit the common mucosal immune system in order to achieve effective respiratory immunity following oral, i.e., gastrointestinal, immunization. The concept of a common mucosal immune system is embodied in the observation that IgA B lymphocytes initially encountering antigen at one mucous membrane can, during their differentiation toward IgA-secreting plasma cells, migrate to other mucous membranes distant from the site of initial antigenic stimulation (Rudzik et al. 1975; Weisz-Carrington et al. 1979). Although antigen is not required for this homing of IgA plasma cell precursors to distant mucous membranes, it has been shown, for example, that antigen applied to the respiratory tract can enhance the number of IgA-bearing, antigen-specific lymphocytes which accumulate in the respiratory tract of orally or enterically primed rodents (Pierce and Cray 1981; Weisz-Carrington et al. 1987). The model system we are using is Sendai virus infection of mice. Sendai virus, a parainfluenza type 1 virus related to human respiratory pathogens, naturally infects mice and can cause a wide spectrum of disease ranging from relatively mild upper respiratory infection to lethal viral pneumonia." @default.
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- W43439608 date "1989-01-01" @default.
- W43439608 modified "2023-10-16" @default.
- W43439608 title "Oral Immunization with Sendai Virus in Mice" @default.
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- W43439608 doi "https://doi.org/10.1007/978-3-642-74529-4_13" @default.
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