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- W4361876721 abstract "<p>Supplementary Figure S1. Involvement of integrin:FAK-signaling in driving ERK activation and proliferation in vivo, related to Figure 1; Supplementary Figure S2. In vitro and in vivo behaviors of various mammary carcinoma cell types, related to Figure 1; Supplementary Figure S3. MoT culture as an in vitro model system to study signaling in DTCs, related to Figures 1 and 2; Supplementary Figure S4. ECM proteins in the lungs that surround recently-extravasated solitary DTCs, related to Figures 1 and 2; Supplementary Figure S5. KSR phosphorylation in nonaggressive/aggressive cancer cell types, related to Figure 2; Supplementary Figure S6. Restoration of ERK activation and proliferation by genetic depletion of Par-1, related to Figure 3; Supplementary Figure S7. Par-1:KSR:Ras/ERK signaling in various cell line models, related to Figure 3; Supplementary Figure S8. Par-1b phosphorylation and its effect on KSR:Ras/ERK signaling, related to Figure 3; Supplementary Figure S9. Function and expression of polarity-regulating proteins, related to Figure 4; Supplementary Figure S10. Distribution of polarity-regulating proteins in 3D conditions, related to Figure 4; Supplementary Figure S11. Restoration of ERK activation and proliferation by genetic depletion of Par-3, related to Figure 5; Supplementary Figure S12. Syndecans as crucial cell-surface sensors for the 3D ECM configuration, related to Figure 6; Supplementary Figure S13. Functional role of syndecans in regulating D2.1 cell behaviors in 3D conditions, related to Figure 6; Supplementary Fig. S14. Genetic inactivation of syndecans and its effect on aggressive cell behaviors, related to Figure 7; Supplementary Fig. S15. Concomitant operations of integrin:FAK-mediated pro-proliferative signaling and syndecan-mediated anti-proliferative signaling in the aggressive D2A1 cells, extended experimental results; Supplementary Fig. S16. Restoration of Par-3 and syndecan-1 expression in the B16F10 melanoma cells, extended experimental results; Supplementary Fig. S17. Evidence for genetic alterations to inactivate syndecan-mediated anti-proliferative signaling machinery in human cancers, extended analyses; Supplementary Fig. S18. Syndecan-mediated anti-proliferative signaling machinery that prevents the outgrowth of solitary DTCs, a summary illustration</p>" @default.
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- W4361876721 date "2023-03-31" @default.
- W4361876721 modified "2023-09-25" @default.
- W4361876721 title "Supplementary Figures (S1-S18) from Syndecan-Mediated Ligation of ECM Proteins Triggers Proliferative Arrest of Disseminated Tumor Cells" @default.
- W4361876721 doi "https://doi.org/10.1158/0008-5472.22422191" @default.
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