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- W4362422169 abstract "<p>FGF19/FGFR4 axis is essential for the effects of H2O2 in HCC cells. (A, B) FGFR4 knockout (FGFR4 KO) and wildtype (WT) MHCC97L cells were treated with 50 μM of H2O2 for 24 hours. Cell viability was determined by MTS assays (A), and cellular release of O2âÂ,¬Â¢- was determined by electrochemical analysis (B). (C, D) FGFR4 knockout MHCC97L cells were transfected with empty vector (EV) or vector containing full-length of FGF19 (FGF19 O/E), followed by treatment with 1 μM of TG for 24 hours. Cell viability was determined by MTS assays (C), and cellular release of O2âÂ,¬Â¢- was determined by electrochemical analysis (D). *P<0.05, N.S. indicates no significant difference.</p>" @default.
- W4362422169 created "2023-04-05" @default.
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- W4362422169 date "2023-03-31" @default.
- W4362422169 modified "2023-09-26" @default.
- W4362422169 title "Supplementary Figure S5 from FGF19 Protects Hepatocellular Carcinoma Cells against Endoplasmic Reticulum Stress via Activation of FGFR4–GSK3β–Nrf2 Signaling" @default.
- W4362422169 doi "https://doi.org/10.1158/0008-5472.22416725.v1" @default.
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