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• W4362558822 abstract "Abstract Schizophrenia (SZ) places a tremendous burden on public health as one of the leading causes of disability and death. SZ patients are more prone to developing obesity than the general population from the clinical practice. The development of obesity frequently causes poor psychiatric outcomes in SZ patients. In turn, maternal obesity during pregnancy has been associated with an increased risk of SZ in offspring, suggesting that these two disorders may have shared neuropathological mechanisms. The gut microbiota is well known to serve as a major regulator of bidirectional interactions between the central nervous system and the gastrointestinal tract. It also plays a critical role in maintaining physical and mental health in humans. Recent studies have shown that the dysbiosis of gut microbiota is intimately associated with the onset of SZ and obesity through shared pathophysiological mechanisms, particularly the stimulation of immune inflammation. Therefore, gut microbiota may serve as a common biological basis for the etiology in both SZ and obesity, and the perturbed gut–brain axis may therefore account for the high prevalence of obesity in patients with SZ. On the basis of these findings, this review provides updated perspectives and intervention approaches on the etiology, prevention, and management of obesity in SZ patients by summarizing the recent findings on the role of gut microbiota in the pathogenesis of SZ and obesity, highlighting the role of gut‐derived inflammation." @default.
• W4362558822 created "2023-04-06" @default.
• W4362558822 creator A5040091526 @default.
• W4362558822 creator A5051552467 @default.
• W4362558822 creator A5051830816 @default.
• W4362558822 creator A5061287221 @default.
• W4362558822 creator A5062290523 @default.
• W4362558822 creator A5075877965 @default.
• W4362558822 date "2023-04-04" @default.
• W4362558822 modified "2023-10-09" @default.
• W4362558822 title "Schizophrenia and obesity: May the gut microbiota serve as a link for the pathogenesis?" @default.
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