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- W4366525210 endingPage "186" @default.
- W4366525210 startingPage "86" @default.
- W4366525210 abstract "Loss of genomic stability in the cell due to defects in the checkpoint of DNA damage, mitotic checkpoint, and telomere maintenance led to increased incidences of base pair alterations. Therefore, that genomic instability plays a critical role in tumor initiation and progression. Tumor progression requires a dynamic tumor/normal exchange in their microenvironment to support tumor growth. The histological alteration seen in the tumor at early stages confirms that the surface between the epithelium and the stroma undergoes progressive disturbance. Tumor progression is also affected by the immune system in which chronic inflammations promote the growth of tumor. Tumor cells experience altered metabolic profiling to support their growth. Cancer cells are characterized by uncontrolled cell division. For that, they utilize glucose as a source of energy to help them grow faster than normal cells. Hence, Glycolysis is a key metabolomics pathway consumed at a high rate during carcinogenesis.<br>" @default.
- W4366525210 created "2023-04-22" @default.
- W4366525210 creator A5009600366 @default.
- W4366525210 creator A5028049373 @default.
- W4366525210 creator A5043655234 @default.
- W4366525210 creator A5046493923 @default.
- W4366525210 date "2023-04-18" @default.
- W4366525210 modified "2023-10-12" @default.
- W4366525210 title "Biology of Cancer" @default.
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