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- W4366606970 abstract "Atopic dermatitis (AD) is a chronic inflammatory skin disease that has been reported as an adverse event during the treatment of inflammatory bowel diseases (IBD) with anti-TNF-α agents. Th2 immune pathway upregulation is key to AD pathogenesis, and increased production of the Th2 cytokines IL-4 and IL-13 substantially contributes to the skin barrier dysfunction, aberrant immune activation, and propagation of inflammation and itch seen in AD.1 It has been theorized that TNF-α blockade may shift the immunologic milieu, favoring Th2 pathway upregulation,2 and personal and familial atopic history may predispose patients to anti-TNF-α–induced AD." @default.
- W4366606970 created "2023-04-23" @default.
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- W4366606970 date "2023-07-01" @default.
- W4366606970 modified "2023-10-18" @default.
- W4366606970 title "Atopic dermatitis induced during anti-TNF-α therapy for inflammatory bowel disease: Potential for Th2 inhibition with dupilumab" @default.
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- W4366606970 doi "https://doi.org/10.1016/j.jaip.2023.03.054" @default.
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