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- W4367675008 endingPage "114827" @default.
- W4367675008 startingPage "114827" @default.
- W4367675008 abstract "Myocardial ischemia-reperfusion injury is a common condition in cardiovascular diseases, and the mechanism of its occurrence involves multiple complex metabolic pathways and signaling pathways. Among these pathways, glucose metabolism and lipid metabolism play important roles in regulating myocardial energy metabolism. Therefore, this article focuses on the roles of glucose metabolism and lipid metabolism in myocardial ischemia-reperfusion injury, including glycolysis, glucose uptake and transport, glycogen metabolism and the pentose phosphate pathway; and triglyceride metabolism, fatty acid uptake and transport, phospholipid metabolism, lipoprotein metabolism, and cholesterol metabolism. Finally, due to the different alterations and development of glucose metabolism and lipid metabolism in myocardial ischemia-reperfusion, there are also complex interregulatory relationships between them. In the future, modulating the equilibrium between glucose metabolism and lipid metabolism in cardiomyocytes and ameliorating aberrations in myocardial energy metabolism represent highly promising novel strategies for addressing myocardial ischemia-reperfusion injury. Therefore, a comprehensive exploration of glycolipid metabolism can offer novel theoretical and clinical insights into the prevention and treatment of myocardial ischemia-reperfusion injury." @default.
- W4367675008 created "2023-05-03" @default.
- W4367675008 creator A5018559115 @default.
- W4367675008 creator A5035076528 @default.
- W4367675008 creator A5040386169 @default.
- W4367675008 creator A5088453668 @default.
- W4367675008 date "2023-07-01" @default.
- W4367675008 modified "2023-10-09" @default.
- W4367675008 title "Abnormalities of glucose and lipid metabolism in myocardial ischemia-reperfusion injury" @default.
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