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- W4368341906 endingPage "449" @default.
- W4368341906 startingPage "435" @default.
- W4368341906 abstract "Nucleic acid sensors survey subcellular compartments for atypical or mislocalized RNA or DNA, ultimately triggering innate immune responses. Retinoic acid-inducible gene-I (RIG-I) is part of the family of cytoplasmic RNA receptors that can detect viruses. A growing literature demonstrates that mammalian RNA polymerase III (Pol III) transcribes certain viral or cellular DNA sequences into immunostimulatory RIG-I ligands, which elicits antiviral or inflammatory responses. Dysregulation of the Pol III-RIG-I sensing axis can lead to human diseases including severe viral infection outcomes, autoimmunity, and tumor progression. Here, we summarize the newly emerging role of viral and host-derived Pol III transcripts in immunity and also highlight recent advances in understanding how mammalian cells prevent unwanted immune activation by these RNAs to maintain homeostasis." @default.
- W4368341906 created "2023-05-05" @default.
- W4368341906 creator A5001156641 @default.
- W4368341906 creator A5016975145 @default.
- W4368341906 creator A5039637095 @default.
- W4368341906 date "2023-06-01" @default.
- W4368341906 modified "2023-10-15" @default.
- W4368341906 title "The RNA polymerase III–RIG-I axis in antiviral immunity and inflammation" @default.
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