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- W4376111305 endingPage "208644" @default.
- W4376111305 startingPage "jcp" @default.
- W4376111305 abstract "Hyperammonaemia (HA) as a consequence of numerous primary or secondary causes, gives rise to clinical manifestations due to its toxic effects on the brain. The neurological consequences broadly reflect the ammonia level, duration and age, with paediatric patients being more susceptible. Drug-induced HA may arise due to either decreased ammonia elimination or increased production. This is associated most frequently with use of valproate and presents a dilemma between ongoing therapeutic need, toxicity and the possibility of an alternative cause. As there is no specific test for drug-induced HA, prompt discussion with a metabolic physician is recommended, as the neurotoxic effects are time-dependent. Specific guidelines for managing drug-induced HA have yet to be published and hence the treatment approach outlined in this review reflects that outlined in relevant urea cycle disorder guidelines." @default.
- W4376111305 created "2023-05-12" @default.
- W4376111305 creator A5059342457 @default.
- W4376111305 creator A5084948649 @default.
- W4376111305 date "2023-05-10" @default.
- W4376111305 modified "2023-09-26" @default.
- W4376111305 title "Drug-induced hyperammonaemia" @default.
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