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- W4377010531 endingPage "112492" @default.
- W4377010531 startingPage "112492" @default.
- W4377010531 abstract "Rhythmic flicker stimulation has gained interest as a treatment for neurodegenerative diseases and as a method for frequency tagging neural activity. Yet, little is known about the way in which flicker-induced synchronization propagates across cortical levels and impacts different cell types. Here, we use Neuropixels to record from the lateral geniculate nucleus (LGN), the primary visual cortex (V1), and CA1 in mice while presenting visual flicker stimuli. LGN neurons show strong phase locking up to 40 Hz, whereas phase locking is substantially weaker in V1 and is absent in CA1. Laminar analyses reveal an attenuation of phase locking at 40 Hz for each processing stage. Gamma-rhythmic flicker predominantly entrains fast-spiking interneurons. Optotagging experiments show that these neurons correspond to either parvalbumin (PV+) or narrow-waveform somatostatin (Sst+) neurons. A computational model can explain the observed differences based on the neurons’ capacitative low-pass filtering properties. In summary, the propagation of synchronized activity and its effect on distinct cell types strongly depend on its frequency." @default.
- W4377010531 created "2023-05-19" @default.
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- W4377010531 creator A5033216984 @default.
- W4377010531 creator A5058168523 @default.
- W4377010531 creator A5084583782 @default.
- W4377010531 date "2023-05-01" @default.
- W4377010531 modified "2023-09-27" @default.
- W4377010531 title "Cell-type-specific propagation of visual flicker" @default.
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- W4377010531 doi "https://doi.org/10.1016/j.celrep.2023.112492" @default.
- W4377010531 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/37195864" @default.
- W4377010531 hasPublicationYear "2023" @default.
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