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- W4377102727 endingPage "114909" @default.
- W4377102727 startingPage "114909" @default.
- W4377102727 abstract "Endometriosis (EM) is characterized by the existence of endometrial mucosa outside the uterine cavity, which causesinfertility, persistent aches, and a decline in women's quality of life. Both hormone therapies and nonhormone therapies, such as NSAIDs, are ineffective, generic categories of EM drugs. Endometriosis is a benign gynecological condition, yet it shares a number of features with cancer cells, including immune evasion, survival, adhesion, invasion, and angiogenesis. Several endometriosis-related signaling pathways are comprehensively reviewed in this article, including E2, NF-κB, MAPK, ERK, PI3K/Akt/mTOR, YAP, Wnt/β‐catenin, Rho/ROCK, TGF‐β, VEGF, NO, iron, cytokines and chemokines. To find and develop novel medications for the treatment of EM, it is essential to implicitly determine the molecular pathways that are disordered during EM development. Additionally, research on the shared pathways between EM and tumors can provide hypotheses or suggestions for endometriosis therapeutic targets." @default.
- W4377102727 created "2023-05-20" @default.
- W4377102727 creator A5002848073 @default.
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- W4377102727 creator A5071732035 @default.
- W4377102727 creator A5080983820 @default.
- W4377102727 date "2023-08-01" @default.
- W4377102727 modified "2023-10-01" @default.
- W4377102727 title "Research advances in endometriosis-related signaling pathways: A review" @default.
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