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- W4377103432 endingPage "8939" @default.
- W4377103432 startingPage "8939" @default.
- W4377103432 abstract "Cyclin-dependent kinases (CDKs) regulate cell division at multiple levels. Aberrant proliferation induced by abnormal cell cycle is a hallmark of cancer. Over the past few decades, several drugs that inhibit CDK activity have been created to stop the development of cancer cells. The third generation of selective CDK4/6 inhibition has proceeded into clinical trials for a range of cancers and is quickly becoming the backbone of contemporary cancer therapy. Non-coding RNAs, or ncRNAs, do not encode proteins. Many studies have demonstrated the involvement of ncRNAs in the regulation of the cell cycle and their abnormal expression in cancer. By interacting with important cell cycle regulators, preclinical studies have demonstrated that ncRNAs may decrease or increase the treatment outcome of CDK4/6 inhibition. As a result, cell cycle-associated ncRNAs may act as predictors of CDK4/6 inhibition efficacy and perhaps present novel candidates for tumor therapy and diagnosis." @default.
- W4377103432 created "2023-05-20" @default.
- W4377103432 creator A5044786101 @default.
- W4377103432 creator A5077445655 @default.
- W4377103432 date "2023-05-18" @default.
- W4377103432 modified "2023-09-28" @default.
- W4377103432 title "Regulation of the Cell Cycle by ncRNAs Affects the Efficiency of CDK4/6 Inhibition" @default.
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