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- W4378593700 abstract "Adriamycin (ADR) is an effective chemotherapy drug for various cancers but has serious side effects. ADR-induced liver damage is a common problem during therapy, but the underlying mechanism remains to be fully understood. In contrast, ADR-induced glomerular damage is well studied in rodents, and sensitivity to ADR-induced nephropathy is because of the R2140C polymorphism of Prkdc gene. To investigate whether strain differences or sensitivity to ADR-induced liver damage are related to Prkdc polymorphism, this study compared the sensitivity to ADR-induced liver damage among C57BL/6J (B6J), B6-PrkdcR2140C, and BALB/c mice. Although B6J exhibits resistance to ADR-induced liver injury, BALB/c and B6-PrkdcR2140C are more susceptible to liver injury, which is exacerbated by the presence of R2140C mutation in PRKDC." @default.
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- W4378593700 date "2023-01-01" @default.
- W4378593700 modified "2023-10-05" @default.
- W4378593700 title "Susceptibility to adriamycin-induced hepatotoxicity in mice depends on PRKDC polymorphism" @default.
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- W4378593700 doi "https://doi.org/10.1292/jvms.23-0115" @default.
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