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- W4380149105 endingPage "131827" @default.
- W4380149105 startingPage "131827" @default.
- W4380149105 abstract "Exposure to PM2.5 is associated with an increased incidence of CNS diseases in humans, as confirmed by numerous epidemiological studies. Animal models have demonstrated that PM2.5 exposure can damage brain tissue, neurodevelopmental issues and neurodegenerative diseases. Both animal and human cell models have identified oxidative stress and inflammation as the primary toxic effects of PM2.5 exposure. However, understanding how PM2.5 modulates neurotoxicity has proven challenging due to its complex and variable composition. This review aims to summarize the detrimental effects of inhaled PM2.5 on the CNS and the limited understanding of its underlying mechanism. It also highlights new frontiers in addressing these issues, such as modern laboratory and computational techniques and chemical reductionism tactics. By utilizing these approaches, we aim to fully elucidate the mechanism of PM2.5-induced neurotoxicity, treat associated diseases, and ultimately eliminate pollution." @default.
- W4380149105 created "2023-06-11" @default.
- W4380149105 creator A5012216290 @default.
- W4380149105 creator A5031890019 @default.
- W4380149105 creator A5040825774 @default.
- W4380149105 creator A5058152911 @default.
- W4380149105 creator A5080513377 @default.
- W4380149105 creator A5089710739 @default.
- W4380149105 date "2023-09-01" @default.
- W4380149105 modified "2023-10-03" @default.
- W4380149105 title "Neurotoxicity of the Air-Borne Particles: From Molecular Events to Human Diseases" @default.
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