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- W4380883114 endingPage "103671" @default.
- W4380883114 startingPage "103671" @default.
- W4380883114 abstract "Rheumatoid arthritis (RA) is an inflammatory, autoimmune and connective-tissue arthropathy. The methotrexate (MTX) and aceclofenac (ACL) combination drug regimen is known to regulate the immunological pathways. Also, RA-elicited inflammation is decreased by the combination drug treatment. ACL and MTX combination treatment has been shown to regulate the signaling pathway controlled by NF-κB and FOXO1. The present manuscript reviews the importance of the combination drug regimen to treat and/or manage RA. The combination drug regimen could affect the Th1/Th17 axis to switch the balance toward the immunoregulatory (Th1) phenotype for establishing immune homeostasis. In conclusion, we propose the study of the immunological signaling pathways in experimental humanized RA mice." @default.
- W4380883114 created "2023-06-17" @default.
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- W4380883114 creator A5074787404 @default.
- W4380883114 date "2023-08-01" @default.
- W4380883114 modified "2023-09-27" @default.
- W4380883114 title "Aceclofenac and methotrexate combination therapy could influence Th1/Th17 axis to modulate rheumatoid-arthritis-induced inflammation" @default.
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- W4380883114 doi "https://doi.org/10.1016/j.drudis.2023.103671" @default.
- W4380883114 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/37330038" @default.
- W4380883114 hasPublicationYear "2023" @default.
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