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- W4381308628 abstract "The gaseous neurotransmitter hydrogen sulfide (H2S) exerts neuroprotective efficacy in the brain via post-translational modification of cysteine residues by sulfhydration, also known as persulfidation. This process is comparable in biological impact to phosphorylation and mediates a variety of signalling events. Unlike conventional neurotransmitters, H2S cannot be stored in vesicles due to its gaseous nature. Instead, it is either locally synthesized or released from endogenous stores. Sulfhydration affords both specific and general neuroprotective effects and is critically diminished in several neurodegenerative disorders. Conversely, some forms of neurodegenerative disease are linked to excessive cellular H2S. Here, we review the signalling roles of H2S across the spectrum of neurodegenerative diseases, including Huntington's disease, Parkinson's disease, Alzheimer's disease, Down syndrome, traumatic brain injury, the ataxias, and amyotrophic lateral sclerosis, as well as neurodegeneration generally associated with ageing." @default.
- W4381308628 created "2023-06-21" @default.
- W4381308628 creator A5006367625 @default.
- W4381308628 creator A5046270567 @default.
- W4381308628 creator A5051421138 @default.
- W4381308628 creator A5070676250 @default.
- W4381308628 creator A5071787930 @default.
- W4381308628 date "2023-07-14" @default.
- W4381308628 modified "2023-10-15" @default.
- W4381308628 title "Hydrogen sulfide signalling in neurodegenerative diseases" @default.
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