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- W4381489205 abstract "To investigate the mechanism by which ginsenoside Rg3 regulates oxidative stress (OS) and inflammation through NF/KB pathway to delay mouse liver injury. This work randomized Balbc mice as four groups: Normal, D-gal, Rg3-L, Rg3-H. Paraffin-embedded liver tissue sections were prepared, later, BAX/BCL-2 protein expression was observed by HE, Sirius red, TUNEL and immunofluorescence to detect apoptotic injury and α-SMA/TGF-β protein expression to detect fibrosis, and liver inflammation-related protein NF-KB was detected. HE and TUNEL staining showed that Rg3 reduced necrotic cells and fibrosis in liver-injured mice, Rg3 increased anti-inflammatory cytokine IL-18 and reduced TNF-α, IL-1β and IL-6 expression. Conclusion: Ginsenoside Rg3 can effectively antagonize D-gal's role in mouse liver injury, and its mechanism may be associated with regulating inflammatory pathway by Rg." @default.
- W4381489205 created "2023-06-22" @default.
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- W4381489205 date "2023-06-21" @default.
- W4381489205 modified "2023-10-17" @default.
- W4381489205 title "Alleviation of D-gal-induced senile liver injury by Rg3, a signature component of red ginseng" @default.
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- W4381489205 doi "https://doi.org/10.18632/aging.204819" @default.
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