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• W4381847854 endingPage "45" @default.
• W4381847854 startingPage "33" @default.
• W4381847854 abstract "COVID-19 is an immune-mediated disease whose pathophysiology uses SAMHD1 tetramerization and cGAS–STING signaling, toll-like receptor 4 (TLR4) cascade, spike protein– inflammasome activation, and neuropilin 1 (NRP1) signaling. Variants of concern, such as SARS-CoV-2 Omicron Subvariants BQ.1, BQ.1.1, BA.4.6, BF.7, BA.2.75.2, and other mutants, have emerged. The longitudinal memory T-cell response to SARS-CoV-2 persists for eight months after symptom onset. Therefore, we must achieve viral clearance to coordinate immune cell reactions. Aspirin, dapsone, and dexamethasone as anticatalysis medicines have been used to treat COVID-19. They are shown to work harmoniously with modulating ILCs. Therefore, it needs to prescribe this immune triad to alleviate the clinical pathologic course and block exacerbation mechanisms due to diverse SARS-CoV-2 variants." @default.
• W4381847854 created "2023-06-25" @default.
• W4381847854 creator A5055775351 @default.
• W4381847854 date "2023-01-01" @default.
• W4381847854 modified "2023-10-18" @default.
• W4381847854 title "Treatment mechanism of immune triad from the repurposing drug against COVID-19" @default.
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