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- W4383738963 abstract "Overexpressed endothelial cell specific molecule-1 (ESM-1) has been identified in various human malignancies, but its expression and function in clear cell renal cell carcinoma (ccRCC) progression are still uncovered. This study explored the critical roles as well as molecular mechanism of ESM-1 in ccRCC progression.The ESM-1 expression in ccRCC tissues and cells was measured using Western blot assay. The function of ESM-1 knockdown in ccRCC cell viability, invasion as well as migration was analysed. Changes in specific proteins were also detected by Western blot analysis.The ESM-1 expression increased in ccRCC tissue samples and cells, which indicated poor prognosis. Moreover, ESM-1 silencing considerably inhibited ccRCC cell growth, invasion and migration in vitro. ESM-1 partially promoted ccRCC development through wingless-type mouse mammary tumour integration site family/beta-catenin (Wnt/β-catenin signalling).ESM-1 acted as an oncogene by influencing the Wnt/β-catenin pathway in ccRCC." @default.
- W4383738963 created "2023-07-11" @default.
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- W4383738963 date "2023-01-01" @default.
- W4383738963 modified "2023-10-16" @default.
- W4383738963 title "ESM-1 Mediates Cell Progression in Clear Cell Renal Cell Carcinoma by Affecting Wnt/β-Catenin Signalling Pathway" @default.
- W4383738963 doi "https://doi.org/10.56434/j.arch.esp.urol.20237604.33" @default.
- W4383738963 hasPubMedId "https://pubmed.ncbi.nlm.nih.gov/37455528" @default.
- W4383738963 hasPublicationYear "2023" @default.
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