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- W4385153914 endingPage "13171" @default.
- W4385153914 startingPage "13148" @default.
- W4385153914 abstract "Excessive production of reactive oxygen species (ROS) in the kidneys is involved in the pathogenesis of kidney diseases, such as acute kidney injury (AKI) and diabetic kidney disease (DKD), and is the main reason for the progression of kidney injury. ROS can easily lead to lipid peroxidation and damage the tubular epithelial cell membrane, proteins and DNA, and other molecules, which can trigger cellular oxidative stress. Effective scavenging of ROS can delay or halt the progression of kidney injury by reducing inflammation and oxidative stress. With the development of nanotechnology and an improved understanding of nanomaterials, more researchers are applying nanomaterials with antioxidant activity to treat kidney injury. This article reviews the detailed mechanism between ROS and kidney injury, as well as the applications of nanozymes with antioxidant effects based on different materials for various kidney injuries. To better guide the applications of antioxidant nanozymes in kidney injury and other inflammatory diseases, at the end of this review we also summarize the aspects of nanozymes that need to be improved. An in-depth understanding of the role played by ROS in the occurrence and progression of kidney injury and the mechanism by which antioxidant nanozymes reduce oxidative stress is conducive to improving the therapeutic effect in kidney injury and inflammation-related diseases." @default.
- W4385153914 created "2023-07-23" @default.
- W4385153914 creator A5001129320 @default.
- W4385153914 creator A5005690754 @default.
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- W4385153914 creator A5065062025 @default.
- W4385153914 creator A5069514005 @default.
- W4385153914 creator A5072211574 @default.
- W4385153914 creator A5085069459 @default.
- W4385153914 creator A5090114526 @default.
- W4385153914 date "2023-01-01" @default.
- W4385153914 modified "2023-10-17" @default.
- W4385153914 title "Antioxidant nanozymes in kidney injury: mechanism and application" @default.
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