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• W4385331312 abstract "Ferroptosis is an iron-dependent cell death process mainly triggered by reactive oxygen species (ROS) and lipid peroxidation. Thioredoxin domain protein 12 (TXNDC12) promotes the development of some tumors; however, its function in tumor ferroptosis remains unclear. In this study, we found that knockdown of TXNDC12 promoted erastin-induced increase in ROS, lipid peroxidation, and Fe2+ levels, and decreased glutathione content. TXNDC12 is involved in ferroptosis by regulating SLC7A11. Further studies showed that TXNDC12 knockdown promoted an erastin-induced decrease in glioma cell viability. Overall, TXNDC12 played a significant role in ferroptosis by modulating SLC7A11 expression. Thus, TXNDC12 and ferroptosis may provide new targets for the treatment of gliomas." @default.
• W4385331312 created "2023-07-29" @default.
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• W4385331312 date "2023-08-10" @default.
• W4385331312 modified "2023-09-26" @default.
• W4385331312 title "TXNDC12 knockdown promotes ferroptosis by modulating SLC7A11 expression in glioma" @default.
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• W4385331312 doi "https://doi.org/10.1111/cts.13604" @default.
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• W4385331312 hasPublicationYear "2023" @default.
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