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- W4385612694 endingPage "83" @default.
- W4385612694 startingPage "72" @default.
- W4385612694 abstract "Mitochondrial transplantation is a promising solution for the heart following ischemia-reperfusion injury due to its capacity to replace damaged mitochondria and restore cardiac function. However, many barriers (such as inadequate mitochondrial internalization, poor survival of transplanted mitochondria, few mitochondria colocalized with cardiac cells) compromise the replacement of injured mitochondria with transplanted mitochondria. Therefore, it is necessary to optimize mitochondrial transplantation therapy to improve clinical effectiveness. By analogy, myocardial ischemia-reperfusion injury is like a withered flower, it needs to absorb enough nutrients to recover and bloom. In this review, we present a comprehensive overview of nutrients (source of exogenous mitochondria and different techniques for mitochondrial isolation), absorption (mitochondrial transplantation approaches, mitochondrial transplantation dose and internalization mechanism), and flowering (the mechanism of mitochondrial transplantation in cardioprotection) for myocardial ischemia-reperfusion injury." @default.
- W4385612694 created "2023-08-06" @default.
- W4385612694 creator A5005391697 @default.
- W4385612694 creator A5012354191 @default.
- W4385612694 creator A5037789032 @default.
- W4385612694 creator A5063852876 @default.
- W4385612694 creator A5087070984 @default.
- W4385612694 creator A5087592842 @default.
- W4385612694 date "2023-09-01" @default.
- W4385612694 modified "2023-10-17" @default.
- W4385612694 title "What can we do to optimize mitochondrial transplantation therapy for myocardial ischemia-reperfusion injury?" @default.
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