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- W4385840669 endingPage "836" @default.
- W4385840669 startingPage "822" @default.
- W4385840669 abstract "Post kala-azar dermal leishmaniasis (PKDL), a heterogeneous dermal sequela of visceral leishmaniasis (VL), is challenging in terms of its etiopathogenesis. Hypopigmentation is a consistent clinical feature in PKDL, but mechanisms contributing to the loss of melanocytes remains poorly defined. Like other hypopigmentary dermatoses – for example, vitiligo, psoriasis, and leprosy – the destruction of melanocytes is likely a multifactorial phenomenon, key players being immune dysregulation and inflammation. This review focuses on immunological mechanisms responsible for the ‘murder’ of melanocytes, prime suspects at the lesional sites being CD8+ T cells and keratinocytes and their criminal tools being proinflammatory cytokines, for example, IFN-γ, IL-6, and TNF-α. Collectively, these may cause decreased secretion of melanocyte growth factors, loss/attenuation of cell adhesion molecules and inflammasome activation, culminating in melanocyte death." @default.
- W4385840669 created "2023-08-16" @default.
- W4385840669 creator A5015534998 @default.
- W4385840669 creator A5034613514 @default.
- W4385840669 creator A5060182390 @default.
- W4385840669 creator A5064768895 @default.
- W4385840669 creator A5077358199 @default.
- W4385840669 date "2023-10-01" @default.
- W4385840669 modified "2023-10-17" @default.
- W4385840669 title "Immune dysregulation and inflammation causing hypopigmentation in post kala-azar dermal leishmaniasis: partners in crime?" @default.
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