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- W4386002254 abstract "Cellular crosstalk plays a vital role in maintaining pancreas homeostasis. Recently, in Cell Metabolism, Basile et al. demonstrated that aberrant upregulation of acinar-cell-specific pancreatic elastase CELA3B within endocrine islets reduces β-cell viability in type 2 diabetes (T2D). This identifies detrimental acinar–β-cell crosstalk as a novel pathogenic mechanism in diabetes." @default.
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- W4386002254 date "2023-11-01" @default.
- W4386002254 modified "2023-10-12" @default.
- W4386002254 title "Abnormal acinar–β-cell crosstalk in type 2 diabetes" @default.
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- W4386002254 doi "https://doi.org/10.1016/j.tem.2023.08.006" @default.
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