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- W4386271595 abstract "Abstract Alzheimer’s disease (AD) is a progressive neurodegenerative disorder and is the most common cause of dementia. The pathogenesis of AD still remains unclear, including two main hypotheses: amyloid cascade and tau hyperphosphorylation. The hallmark neuropathological changes of AD are extracellular deposits of amyloid-β (Aβ) plaques and intracellular neurofibrillary tangles (NFTs). Endocytosis plays an important role in a number of cellular processes including communication with the extracellular environment, nutrient uptake, and signaling by the cell surface receptors. Based on the results of genetic and biochemical studies, there is a link between neuronal endosomal function and AD pathology. Taking this into account, we can state that in the results of previous research, endolysosomal abnormality is an important cause of neuronal lesions in the brain. Endocytosis is a central pathway involved in the regulation of the degradation of amyloidogenic components. The results of the studies suggest that a correlation between alteration in the endocytosis process and associated protein expression progresses AD. In this article, we discuss the current knowledge about endosomal abnormalities in AD." @default.
- W4386271595 created "2023-08-31" @default.
- W4386271595 creator A5002054623 @default.
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- W4386271595 creator A5052973150 @default.
- W4386271595 creator A5055213999 @default.
- W4386271595 creator A5059511347 @default.
- W4386271595 creator A5062059974 @default.
- W4386271595 date "2023-08-30" @default.
- W4386271595 modified "2023-09-30" @default.
- W4386271595 title "Endocytosis and Alzheimer’s disease" @default.
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