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- W4386294890 abstract "<p>Global methylation is associated with genomic stability and tumor subtype. <b>A,</b> Overall methylation profiles at repetitive regions, as well as their upstream and downstream 2 kb regions in normal epithelial cell lines, IMU, KRT subtypes, and HPV(−) tumors. <b>B,</b> Average methylation at repetitive elements (Alu, L1, and L2) by subtype. <b>C,</b> Boxplots of genome-wide average methylation in normal cell types and subtypes/HPV(−) HNSCC samples of our cohort. <b>D,</b> Average cancer-specific methylation grouped by HPV(+) HNSCC subtypes (IMU and KRT) and HPV(−) HNSCC. Cancer-specific methylation levels derived from EPIC and MethylCIBERSORT approaches are shown separately. <b>E,</b> Correlation between genomic instability and cancer-specific methylation of the 36 HNSCC samples, denoted by different colors according to their subtypes. The Pearson correlation (<i>R</i>) and correlation test <i>P</i> value are shown on top right.</p>" @default.
- W4386294890 created "2023-08-31" @default.
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- W4386294890 date "2023-08-30" @default.
- W4386294890 modified "2023-10-16" @default.
- W4386294890 title "FIGURE 2 from Whole-genome CpG-resolution DNA Methylation Profiling of HNSCC Reveals Distinct Mechanisms of Carcinogenesis for Fine-scale HPV+ Cancer Subtypes" @default.
- W4386294890 doi "https://doi.org/10.1158/2767-9764.24058576" @default.
- W4386294890 hasPublicationYear "2023" @default.
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