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- W4386822260 abstract "We have read with great interest the article by Nair et al[1] in which they explain the presence of two subphenotypes of acute respiratory distress syndrome (ARDS) secondary to severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) through serum markers of systemic inflammation such as ferritin (ferritin), serum lactate dehydrogenase (LDH) and C-reactive protein (CRP), which are associated with worse results in terms of days of stay in the intensive care unit (ICU), days of mechanical ventilation, and higher mortality; interleukin-6 (IL-6) is the proinflammatory cytokine involved in the cascade of systemic damage. Therefore we share our experience and research on the other side of the world: under the premise that elevated serum levels of IL-6[2] favor non-osmotic secretion of antidiuretic hormone (ADH) with the consequent presence of hyponatremia this electrolyte disturbance could be another marker of severity and poor prognosis; therefore, SARS-CoV-2 would be the etiological agent in which the IL-6 released is recognized as the main inflammatory mediator of the acute phase with hematological, immunological, endocrinological, and metabolic effects.[3]" @default.
- W4386822260 created "2023-09-19" @default.
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- W4386822260 date "2023-09-18" @default.
- W4386822260 modified "2023-09-26" @default.
- W4386822260 title "Hyponatremia: A Marker of Inflammation for COVID-19" @default.
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- W4386822260 doi "https://doi.org/10.1055/s-0043-1773808" @default.
- W4386822260 hasPublicationYear "2023" @default.
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