FRINK Linked Data Fragments server

Matches in SemOpenAlex for { <https://semopenalex.org/work/W4386879881> ?p ?o ?g. }

• W4386879881 endingPage "e20363" @default.
• W4386879881 startingPage "e20363" @default.
• W4386879881 abstract "Ischemia/reperfusion (I/R) injury is an inevitable dilemma when previously ischemic multiple organs and tissues are returned to a state of blood flow, with confirming a critical role of ferroptosis in molecular, pathway mechanisms, subcellular structure. Discovering the potential relationship may provide useful approaches for the clinical treatment and prognosis of the pathophysiological status of IRI. Therefore, a comprehensive visualization and scientometric analysis were conducted to systematically summarize and discuss the ferroptosis in ischemia reperfusion injury research to demonstrate directions for scholars in this field.We retrieved all publications focusing on I/R injury and ferroptosis from the Web of Science Core Collection (WoSCC), published from 2013 to October 2022. Next, scientometric analysis of different items was performed using various bibliometrics softwares to explore the annual trends, countries/regions, institutions, journals, authors and their multi-dimensional relationship pointing to current hotspots and future advancement in this field.We included a total of 421 English articles in set timespan. The number of publications increased steadily annually. China produced the highest number of publications, followed by the United States. Most publications were from Central South University, followed by Sichuan University and Wuhan University. The most authoritative academic journal was Oxidative Medicine and Cellular Longevity. Cell occupied the first rank of co-cited journal list. Andreas Linkermann and Scott J Dixon may have the highest influence in this intersected field with the highest number of citations and co-cited references respectively. The essential biological reactions such as oxidative stress response, lipid peroxidation metabolism, anti-inflammmatory and pro-inflammatory procedure, and related molecular pathways were knowledge base and current hotspots. Molecules pathways exploration, effective inhibition of I/R injury and promising strategy of improving allografts may become future trends and focuses.Research on ferroptosis in I/R injury had aroused great interest recently. This first bibliometric study comprehensively analyzed the research landscape of ferroptosis and I/R injury, and also provided a reliable reference for related scholars to facilitate further advancement in this field." @default.
• W4386879881 created "2023-09-21" @default.
• W4386879881 creator A5003706139 @default.
• W4386879881 creator A5006157945 @default.
• W4386879881 creator A5007139780 @default.
• W4386879881 creator A5019084328 @default.
• W4386879881 creator A5022034604 @default.
• W4386879881 creator A5026473596 @default.
• W4386879881 date "2023-10-01" @default.
• W4386879881 modified "2023-10-04" @default.
• W4386879881 title "Knowledge mapping and emerging trends of ferroptosis in ischemia reperfusion injury research: A bibliometric analysis (2013–2022)" @default.
• W4386879881 cites W1986283558 @default.
• W4386879881 cites W1991997670 @default.
• W4386879881 cites W1993613360 @default.
• W4386879881 cites W1997183854 @default.
• W4386879881 cites W2002490399 @default.
• W4386879881 cites W2011538447 @default.
• W4386879881 cites W2025161139 @default.
• W4386879881 cites W2035898183 @default.
• W4386879881 cites W2045171661 @default.
• W4386879881 cites W2057632732 @default.
• W4386879881 cites W2062110054 @default.
• W4386879881 cites W2067107606 @default.
• W4386879881 cites W2075330571 @default.
• W4386879881 cites W2135028945 @default.
• W4386879881 cites W2150146658 @default.
• W4386879881 cites W2163005196 @default.
• W4386879881 cites W2163294546 @default.
• W4386879881 cites W2180777157 @default.
• W4386879881 cites W2265062179 @default.
• W4386879881 cites W2317629072 @default.
• W4386879881 cites W2781994781 @default.
• W4386879881 cites W2788640168 @default.
• W4386879881 cites W2792968897 @default.
• W4386879881 cites W2887586296 @default.
• W4386879881 cites W2911996561 @default.
• W4386879881 cites W2914574872 @default.
• W4386879881 cites W2916144846 @default.
• W4386879881 cites W2928467655 @default.
• W4386879881 cites W2953282350 @default.
• W4386879881 cites W2961268319 @default.
• W4386879881 cites W2982991142 @default.
• W4386879881 cites W3000699351 @default.
• W4386879881 cites W3005192045 @default.
• W4386879881 cites W3016555862 @default.
• W4386879881 cites W3037902450 @default.
• W4386879881 cites W3044388227 @default.
• W4386879881 cites W3062364896 @default.
• W4386879881 cites W3092432496 @default.
• W4386879881 cites W3094759632 @default.
• W4386879881 cites W3096449942 @default.
• W4386879881 cites W3105907500 @default.
• W4386879881 cites W3106504926 @default.
• W4386879881 cites W3107334044 @default.
• W4386879881 cites W3119155918 @default.
• W4386879881 cites W3123922203 @default.
• W4386879881 cites W3149705980 @default.
• W4386879881 cites W3159603454 @default.
• W4386879881 cites W3169250234 @default.
• W4386879881 cites W3171018860 @default.
• W4386879881 cites W3188638889 @default.
• W4386879881 cites W3195829662 @default.
• W4386879881 cites W3197759514 @default.
• W4386879881 cites W3212931987 @default.
• W4386879881 cites W3215658413 @default.
• W4386879881 cites W3217380155 @default.
• W4386879881 cites W323406638 @default.
• W4386879881 cites W4206176457 @default.
• W4386879881 cites W4210557083 @default.
• W4386879881 cites W4213041930 @default.
• W4386879881 cites W4213095840 @default.
• W4386879881 cites W4214725121 @default.
• W4386879881 cites W4214872440 @default.
• W4386879881 cites W4220782105 @default.
• W4386879881 cites W4221092109 @default.
• W4386879881 cites W4223423231 @default.
• W4386879881 cites W4223567523 @default.
• W4386879881 cites W4224212502 @default.
• W4386879881 cites W4224216903 @default.
• W4386879881 cites W4224273882 @default.
• W4386879881 cites W4280613393 @default.
• W4386879881 cites W4281388265 @default.
• W4386879881 cites W4281478732 @default.
• W4386879881 cites W4281744542 @default.
• W4386879881 cites W4282009814 @default.
• W4386879881 cites W4282837350 @default.
• W4386879881 cites W4290695027 @default.
• W4386879881 cites W4290755668 @default.
• W4386879881 cites W4291017337 @default.
• W4386879881 cites W4293004208 @default.
• W4386879881 cites W4294176139 @default.
• W4386879881 cites W4296027835 @default.
• W4386879881 cites W4296329144 @default.
• W4386879881 cites W4306761770 @default.
• W4386879881 cites W4306809501 @default.
• W4386879881 cites W4307427737 @default.
• W4386879881 cites W4308202083 @default.
• W4386879881 cites W4309822006 @default.

• next