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- W4386996810 endingPage "14494" @default.
- W4386996810 startingPage "14494" @default.
- W4386996810 abstract "The formation of atherosclerotic plaques is one of the main sources of cardiovascular disease. In addition to known risk factors such as dyslipidemia, diabetes, obesity, and hypertension, endothelial dysfunction has been shown to play a key role in the formation and progression of atherosclerosis. Peroxisome proliferator-activated receptor-gamma (PPARγ), a transcription factor belonging to the steroid superfamily, is expressed in the aorta and plays a critical role in protecting endothelial function. It thereby serves as a target for treating both diabetes and atherosclerosis. Although many studies have examined endothelial cell disorders in atherosclerosis, the role of PPARγ in endothelial dysfunction is still not well understood. In this review, we summarize the possible mechanisms of action behind PPARγ regulatory compounds and post-translational modifications (PTMs) of PPARγ in the control of endothelial function. We also explore the potential use of endothelial PPARγ-targeted agents in the prevention and treatment of atherosclerosis." @default.
- W4386996810 created "2023-09-25" @default.
- W4386996810 creator A5004676297 @default.
- W4386996810 creator A5037152409 @default.
- W4386996810 creator A5055543088 @default.
- W4386996810 date "2023-09-24" @default.
- W4386996810 modified "2023-10-17" @default.
- W4386996810 title "PPARγ in Atherosclerotic Endothelial Dysfunction: Regulatory Compounds and PTMs" @default.
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