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- W4387204474 abstract "Inflammation: A better model of sepsis The surgical removal of fat deposits in mice results in a more realistic animal model of the sepsis resulting from severe infection. For decades, researchers have studied this potentially fatal inflammatory process by introducing punctures into the mouse intestinal wall. The usefulness of this model can be undermined if abscesses form to physically confine the immune response that drives sepsis, but Shuhei Niiyama and colleagues at Japan's Kurume University School of Medicine have devised a potential solution. They determined that pockets of fatty tissue around the intestine contribute to abscess formation, and that removing these deposits considerably promotes the onset of sepsis after intestinal puncture. These results support prior evidence that fat cells can generate signals that restrain inflammation, and could lead to more effective studies of drugs for treating this toxic outcome of infection." @default.
- W4387204474 created "2023-09-30" @default.
- W4387204474 date "2016-02-01" @default.
- W4387204474 modified "2023-09-30" @default.
- W4387204474 doi "https://doi.org/10.1002/cti2.2016.5.issue-2" @default.
- W4387204474 hasPublicationYear "2016" @default.
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