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- W44172843 abstract "Severe sepsis triggers clotting, diminishes the activity of natural anticoagulant mechanisms, and impairs the fibrinolytic system. Augmented interactions between inflammation and coagulation can give rise to a vicious cycle, eventually leading to dramatic events such as manifested in severe sepsis and DIC. Unraveling the role of coagulation and inflammation in sepsis will pave the way for new treatment targets in sepsis that can modify the excessive activation of these systems. At present it remains unclear whether anticoagulant therapy improves survival in severe sepsis; in addition, it remains uncertain whether the beneficial effect of recombinant human APC derives from its anticoagulant properties. These issues will be clarified as our understanding of the interplay between coagulation and inflammation during sepsis improves further in the very near future." @default.
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- W44172843 date "2007-01-01" @default.
- W44172843 modified "2023-09-26" @default.
- W44172843 title "Coagulation in Sepsis" @default.
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- W44172843 doi "https://doi.org/10.1007/3-540-30328-6_19" @default.
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