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- W46932380 endingPage "134" @default.
- W46932380 startingPage "91" @default.
- W46932380 abstract "Helicobacter pylori is a spiral-shaped, flagellated, microaerophilic Gram-negative bacterium that colonizes the gastric epithelium of c.50% of humans (Blaser & Berg 2001). These organisms elicit a strong immune response that does not resolve the infection; once acquired, bacteria persist for a lifetime in the absence of antibiotic treatment. All persons infected with H. pylori have gastritis and 20%–30% will develop severe disease that includes gastric and duodenal ulcers, gastric adenocarcinoma or MALT lymphoma (Covacci et al. 1999). A characteristic feature of H. pylori infection is the massive recruitment of phagocytes (particularly neutrophils) to the gastric mucosa (Allen 2000). Of interest here is the fact that H. pylori survives for years in a phagocyte-rich environment, and a growing body of data demonstrates that these organisms modulate the host inflammatory response and phagocyte function. By this mechanism H. pylori evades phagocytic killing and promotes host tissue damage." @default.
- W46932380 created "2016-06-24" @default.
- W46932380 creator A5052550075 @default.
- W46932380 date "2006-09-07" @default.
- W46932380 modified "2023-09-27" @default.
- W46932380 title "Life, death, and inflammation: manipulation of phagocyte function by <i>Helicobacter pylori</i>" @default.
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