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- W4785151 abstract "Detailed studies of the mechanisms of complement attack at bacterial surfaces have led to the discovery that different groups of bacteria have evolved different mechanisms to avoid complement-mediated attack. The enterobacteriaceae have evolved a mechanism of bacterial resistance in which all components bind to the bacterial surface. When the membrane attack sequence is completed the complex does not integrate successfully into the outer membrane and is shed from the bacterial surface. In addition certain types of lipopolysaccharides bind C3 relatively poorly, thus reducing lytic potential. Gonococci on the other hand appear to resist complement attack by binding components to surface structures that effectively preclude insertions into vital membrane sites. The role of bactericidal antibody is also considered. In many cases antibody appears to direct complement attack. Although relatively the same numbers of components are deposited on bacterial surfaces, in the presence of bactericidal antibody they mediate lysis. Some antibodies may direct the attack sequence to be deposited at sites where lysis is precluded." @default.
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- W4785151 date "1986-01-01" @default.
- W4785151 modified "2023-10-18" @default.
- W4785151 title "Molecular Mechanisms in the Antibacterial Action of Complement" @default.
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- W4785151 doi "https://doi.org/10.1016/b978-0-12-174685-8.50031-7" @default.
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